The researchers conclude, "P. gingivalis modifies its lipid A structure in order to evade host defenses and establish chronic infection leading to persistent systemic low-grade inflammation." They go on to state that "uniquely among gram-negative pathogens, P. gingivalis evasion of TLR4-mediated host immunity results in progression of inflammation at a site that is distant from local infection by gaining access to the vasculature."I'm expecting some kind of denial from the AHA in 5, 4, 3...
Friday, July 11, 2014
It's Indisputable
On the link between periodontitis and atherosclerosis