The researchers found substantial increases in amyloid beta peptide pathology -- a hallmark of Alzheimer's disease -- in the brain cortex and hippocampus concurrent with diabetes. They also found significant amyloid beta pathology in the retina and by contrast, when diabetes is not present, no observable pathology was detected in either the brain or the retina.This is how I am beginning to think it works-
APOE4 compromises cell immunity to HSV1, and susceptible individuals are infected.
Herpes infection impairs the beta-amyloid pathway, which controls insulin processing.
Affected individuals with comorbid Hyperinsulinemia experience insulin resistance compounded by beta-amyloid accumulation.
